I'm sure you have a lot less AA in your cells and more Mead Acid than normal people eating high-PUFA oils. But you are moving the goal-posts. You said you had "eliminated"AA in your cells. I agree that you will most likely show up as EFAD, based on the triene:tetraene (Mead:AA) ratio. But that does not mean you have no AA. You might have more AA in your cells than Mead Acid and still be deemed EFAD. I think that criteria for EFAD is flawed, any way.
I've noticed similar benefits from restricting PUFAs. My cuts stop bleeding faster and form a soft scab with no itchiness or inflammation. I noticed years ago while using flax oil that it suppressed my immune system. I got sick easily when I was exposed to cold. I just wash my hair with water, run my hands through it, and comb it gently.
There are some ways that I think we could deplete AA, such as subjecting ourselves to mild stresses. Example: periodic fasting, cold showers, hot-and-cold (contrast) showers, sleep deprivation, high-intensity exercise, ketosis, etc.
What about the argument that people are "growing too fast" nowadays because of PUFAs and junk fats? This book I am reading cites a study (Tanner JM. 1973. Trend toward earlier menarche in London, Oslo, Copenhagen, the Netherlands and Hungary. Nature 243: 75-76). Norway and Finland both had female menarche around 16.5-17.0 years old, back in the early to mid-1800s. By 1970, they were both down near 13 years. They were probably eating a lot of dairy back then and now they are eating PUFAs and junk fats.
Their consumption of sugar and refined carbs was probably less in the 1800s than it is today, going by the trends in other industrialized countries. These two factors combined would accelerate lipid peroxidation, vitamin E depletion, and all of the chronic inflammatory disease we see today. |
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